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Obesity as a Neuro-Metabolic Disease Key mechanisms include: Neuroendocrine dysregulation of appetite and satiety Metabolic adaptation defending higher weight Environmental and behavioral feedback loops Strong relapse tendency requiring long-term care Lifestyle alone is rarely sufficient for durable control

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Efficacy magnitude Clinical trials show GLP-1 agonists produce substantial HbA1c reductions (typically 1.01.5% depending on agent and dose) and weight loss (515% of body weight with higher doses)

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Another microarray assay compared early gene expression responses in A