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Class 1 ferroptosis inducers, including erastin, imidazole ketone erastin, sulfasalazine, sorafenib, and HG106, directly inhibit xCT activity [92,93] and induce ferroptosis by preventing cystine uptake and depleting cysteine or GSH, thereby inducing lipid peroxidation and cell death [15,49]
Oral glutathione has poor bioavailability it is largely broken down in the gastrointestinal tract before reaching systemic circulation
However, there is always more than one transporter capable of moving one specific AA from one side to the other
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