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GLP1/E therapy also elicited specific changes in the hypothalamic proteomic profile in PNA mice, upregulating proteins involved in the metabolism of non-coding RNA, signaling of nuclear receptor and estrogen-dependent gene expression and downregulating pathways involved in apoptosis, signaling by insulin receptor and cellular responses to starvation
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Its glucose-lowering and insulinotropic efficacy was superior to that of selective GLP-1 receptor agonists in diet-induced obese and leptin receptor-deficient mice, as well as in monkeys and humans [74]
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