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The potential mechanism responsible for these differences may involve a switch in the release of immunostimulatory danger/damage-associated molecular patterns (DAMPs) (e.g., ATP and reduced HMGB1 [high mobility group box 1]) to immune-suppressive DAMPs (e.g., oxidized HMGB1 or PGE2 [prostaglandin E2]) [198, 199]

Recovery speed improvements typically show up in weeks three to four