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The real issue often lies deeper, at the cellular level

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In contrast, mutant TP53 has been proved to drive the glycolysis by activating the RhoA/ROCK/GLUT1 signaling cascade (164), repress the catabolic activities, such as fatty acid oxidation (FAO), by inhibiting 5-AMP-activated protein kinase (AMPK) pathways, and enhance the anabolic processes, such as enhanced fatty acid synthesis (174)

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For a long time, the answer was a frustrating "probably not." But then, a groundbreaking study from Penn State University changed everything, providing the first solid proof we'd all been waiting for