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GHK-Cu is non-hormonal, working instead through tissue repair and anti-inflammatory activity

What distinguishes Epithalon from other longevity-associated molecules under preclinical investigation is the specificity of its proposed mechanism: upregulation of hTERT (human telomerase reverse transcriptase) gene expression, the catalytic subunit that constitutes the rate-limiting determinant of telomerase activity

[15] [30] Resolves major vessel occlusions and ischemia-reperfusion injury via cytoprotection and collateral pathway activation

Oxidative stress and beta amyloid in Alzheimers disease: which comes first

PubMed laboratory research record [PMID: 31266512] PubMed record related to this compound for laboratory literature review