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In skeletal muscle this pathway can support satellite cell activation and possible hyperplasia (creation of new muscle fibers)
through the combination of sulfydryl and free radicals in vivo, the free radicals can be directly reduced and converted into easily metabolized acid substances, so that the excretion of the free radicals is accelerated, and the damage of the free radicals to important organs can be resisted
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Emerging evidence underscores the importance of integrin-mediated mechanotransduction in regulating cardiac pathophysiology under conditions of mechanical stress overload
