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Hyperglycemia-induced oxidative stress and inflammation activate intrinsic and extrinsic apoptotic pathways, leading to programmed cell death of neurons and Schwann cells
CLIC1 undergoes a large conformation change upon oxidation
Waiting for diagnosis carries a high risk, while injecting B12 carries no risk
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ROS Scavenging and Gene Regulation: Oxidative stress upregulates the Microphthalmia-associated Transcription Factor (MITF), which acts as the primary on-switch for melanin production [5]
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