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The investigation of ferroptosis in ischemic stroke is fundamentally shaped by the experimental context, where a stark disparity exists between reductionist in vitro systems and complex in vivo models, a critical consideration for interpreting the literature and guiding translational efforts

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however, our data show that BACH1 stabilization promotes angiogenesis, which may be attributed to cell type specificity or context-dependent downstream targets (e.g., PDGFR instead of HO-1) [17, 18]

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