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GHK-Cu se prvenstveno prouava u modelima bakarne signalizacije, fibroblasta, kolagena i ekstracelularnog matriksa, dok BPC-157 ima drugaiju strukturu i iri prekliniki istraivaki profil

Rather than directly supplying NAD+ precursors (like NMN or NR), it works by blocking an enzyme that consumes nicotinamide, theoretically preserving the substrate pool for endogenous NAD+ production

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Detailed biophysical studies (X-ray crystallography, EPR, NMR) elucidated that in GHK-Cu, the Cu(II) ion is coordinated by the imidazole side chain nitrogen of histidine, the alpha-amino nitrogen of glycine, and the deprotonated amide nitrogen of the glycine-histidine bond

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