Fix the deficiency fix the absorption
This doesnt happen with injectable nutrition
Haemolysis in individuals with G6PD deficiency has been reported to follow therapy with a range of anti-malarial drugs (8-aminoquinolines, including primaquine, tafenoquine and pamaquine), sulphones (dapsone), sulphonamides (such as sulphanilamide, sulphamethoxazole, and mafenide), analgesics (such as aspirin, phenazopyridine, and acetanilide), non-sulpha antibiotics (nalidixic acid, nitrofurantoin, isoniazid, and furazolidone), methylene blue and naphthalene [13]
DPP-4 inhibitors did not lower the risk of any cardiorenal outcome when compared with placebo and were associated with higher risks of MACE, HHF, and renal outcome when compared with the other two drug classes
In mice, CHAC1/BOTCH promotes neuronal differentiation by inhibiting NOTCH1 and both its depletion and overproduction severely disrupt neurogenesis ( CHAC1/BOTCH in zebrafish leads to severe defects in brain, heart and myotome development, resulting in embryonic lethality, highlighting its conserved and essential role (Yadav et al., 2019)