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Third, as we will detail below (see Potential Pathogenic Mechanisms of Hyperhomocysteinemia), our own work (Durand et al, 1997a) and the work of Lentz et al (1996) and Bellamy et al (1998) give experimental evidence that moderate hyperhomocysteinemia, which leads to vascular dysfunction, might increase the cardiovascular risk independently of other risk factors

[4] [6] The proposed mechanism involves rapid weight loss leading to changes in bile composition and gallbladder motility

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[5] During deep sleep, the body engages in the repair and regeneration process, and Glutathione plays a vital role in this cellular repair

However, results are mixed, but the potential is encouraging