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pneumoniae and is dependent on the enzymatic activities of SodA and TpxD, which function to neutralize superoxide radicals and hydrogen peroxide, respectively [3, 8]

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An oxidant/antioxidant imbalance characterized by oxidative stress and low GSH levels is involved in the pathogenesis of idiopathic pulmonary fibrosis, since data show marked GSH deficiency in the lower respiratory tract of those patients (127)

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