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The most frequent clinical manifestations of G6PD deficiency are neonatal jaundice and acute hemolytic anemia, which are initiated by exogenous oxidative agents, including drugs, infection or ingestion of fava beans (4,6)

While it is too early to call an end to compounding, potential intervention by the FDA and DOJ could support the branded GLP-1 market, Flynn says

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Combination approaches that pair GSH depletion with ferroptosis induction, immunotherapy, or chemotherapy may help overcome resistance, while standardized manufacturing and rigorous toxicity evaluation will be critical for regulatory approval